While users on Windows Phone have been enjoying the ability to post to Instagram both unofficially (Instagraph, Instance, etc.) and officially (Hipstamatic Oggl), there has been one complicated and vexing issue haunting people: tags.
The concern can be best explained as thus: when users post using third party apps from Windows Phone, those images are not searchable if they use a hashtag. For example, if you publish using Instance with the hashtag ?#wp8? in theory when you use the search key to find other photos with ?#wp8?, you should see your latest post indexed. However, this is not the case as your image will be hidden.
Rudy Huyn, the developer working on the forthcoming 6tagram app has posted an article via his blog explaining the situation. The news is not very good and highlights the continued cat and mouse game between Windows Phone developers and the services they are trying to make apps for.
Background
A test of hashtags from a flagged account
When Instagram instituted their new security fixes a few weeks ago to reduce spam-bots and unauthorized breaches, the changes also had the side effect of causing interference with third party apps on Windows Phone. The first round of that was any image posted was automatically deleted after about fifteen seconds. That was later improved so that your images stuck, but they were then marked ?private? meaning only your followers could see it. Finally, the strings were loosened once more and again, your images were visible to everyone.
At the time, that was great news. However, it is now becoming clear that all is still not right. Images posted with third party apps with a hashtag won?t have those images indexed to be searchable on the Instagram network. That means if we posted a photo of an unreleased Windows Phone with the hashtag #wp8, the only people who would see it were those who follow us (or via other social networks e.g. Twitter, Facebook, if we chose to share it there as well).
Hashtags, like on Twitter, are an important tool for discovery on the photo sharing network. Without them it makes it hard to increase your follower count, as it will only be by word of mouth or other social networks that new users can see your contribution.?
Who?s affected?
Huyn's 6tagram "warning" has ruffled some feathers amongst?developers
The consensus seems to be that anyone who posted via Instance, Instagraph (rarer, though still possible) and InstaPic (for Windows 8) now have their accounts flagged for hashtag indexing. Even users of the 6tagram beta (version 12 and earlier) will have been marked by Instagram as potentially being spam.
Our account at daniel_rubino has been identified as offending, making our hashtags useless. That account was created on Android, first posted using Instagraph, with the bulk of the uploaded images via Instance.
Can you be removed from the blacklist?
The forthcoming Instance v2.0 may solve this problem
Yes, in theory, but it gets complicated. In short, if you stop using those offending apps and logout from them, after around seven days your account may be normalized, but that is merely speculation at this point.
The flipside to this is that currently none of the apps in question (except 6tagram beta), actually send the proper logout request to Instagram. Instead, they only wipe the login credentials and cookies locally on the device. Until those developers add in that ability, users simply won?t have a method to get off of the blacklist anytime soon.
Is there a solution for future users?
Yes, so far at least with 6tagram beta 13 and going forward, the problem has been resolved thru a hack. That means when 6tagram hits the Windows Phone Store officially for public release, new users should be ok. Likewise if you never posted to Instagram or have only used the 6tagram beta.
It?s not clear if the developers behind Instagraph, InstaPic or Instance will have similar solutions, though Daniel Gary (developer behind Instance) is aware of the problem and may have a solution for his customers too in his upcoming Instance version 2.0.
The price of not being official and unintented consequences
At this point, Windows Phone users are still being punished for not having an official app from Instagram. While these third party options offer a way in, despite their well-polished design they are still second tier and will suffer these impediments in development.
While it?s our understanding that Microsoft and Instagram are still having talks and an app from Microsoft is ready (complete with lockscreen support and notifications) there is no evidence that a release is impending due to the lack of an agreement between the two companies. Indeed, Microsoft could be angling for deep-level access akin to Facebook and LinkedIn for its users--but that process could take time.
For now, Windows Phone users will just have to get by and put up with these difficulties.
[unable to retrieve full-text content]As the administration considers approval of the much-debated Keystone XL pipeline, cleanup efforts in two communities portend the potential hazards of transporting heavy Canadian crude.
Roku introduced the "Play on Roku" feature to broadcast still images and music from mobile devices to its set-top box late last year, and now it's iOS app has added video support to the mix. Currently available only for user-recorded videos it's no AirPlay-replacement, but in the crowded media streamer market any extra functionality is welcome. As is the case with many recently introduced features on the platform, you'll need some of Roku's second gen or newer hardware to take advantage, but if you're properly equipped then just grab the update at the link below.
This May 22, 2009 photo shows the sign for Wells Fargo banks in Woodbury, Minnesota. (AFP | KAREN BLEIER)
Wells Fargo announced Wednesday the termination of 118 positions in its consumer-mortgage-loan-processing section in metro Denver.
Cristie Drumm, Wells Fargo spokeswoman for Colorado, said the employees were informed of the decision Wednesday morning.
Drumm said the decision was the result of slower demand for mortgage refinancing than Wells Fargo experienced throughout 2012 and early 2013.
Nationwide, she said, 763 mortgage-loan-processing jobs were being eliminated.
Drumm emphasized that Wells Fargo is committed to retaining as many of the employees ? who were given 60 days' notice ? as it can.
"What we would do, we would meet with each of the team members individually, talk with them about what they might like to do, what other positions they might like to have, the different skills they have and see if we can match them up with an open position," said Drumm.
Drumm said the positions could be anywhere in the United States.
Howard Pankratz: 303-954-1939, hpankratz@denverpost.com or twitter.com/howardpankratz
This summer, ten small libraries mysteriously appeared throughout New York City's Lower East Side and East Village. But who paid for them? Who designed them? And what was the point? In a short film published today, the creators finally answer our many questions about how the Little Free Library came to be.
Be happy: Your genes may thank you for itPublic release date: 29-Jul-2013 [ | E-mail | Share ]
Contact: Mark Wheeler mwheeler@mednet.ucla.edu 310-794-2265 University of California - Los Angeles
But different types of happiness have different effects, UCLA study shows
A good state of mind that is, your happiness affects your genes, scientists say. In the first study of its kind, researchers from UCLA's Cousins Center for Psychoneuroimmunology and the University of North Carolina examined how positive psychology impacts human gene expression.
What they found is that different types of happiness have surprisingly different effects on the human genome.
People who have high levels of what is known as eudaimonic well-being the kind of happiness that comes from having a deep sense of purpose and meaning in life (think Mother Teresa) showed very favorable gene-expression profiles in their immune cells. They had low levels of inflammatory gene expression and strong expression of antiviral and antibody genes.
However, people who had relatively high levels of hedonic well-being the type of happiness that comes from consummatory self-gratification (think most celebrities) actually showed just the opposite. They had an adverse expression profile involving high inflammation and low antiviral and antibody gene expression.
The report appears in the current online edition of the journal Proceedings of the National Academy of Sciences.
For the last 10 years, Steven Cole, a UCLA professor of medicine and a member of the UCLA Cousins Center, and his colleagues, including first author Barbara L. Fredrickson at the University of North Carolina, have been examining how the human genome responds to stress, misery, fear and all kinds of negative psychology.
In this study, though, the researchers asked how the human genome might respond to positive psychology. Is it just the opposite of stress and misery, or does positive well-being activate a different kind of gene expression program?
The researchers examined the biological implications of both hedonic and eudaimonic well-being through the lens of the human genome, a system of some 21,000 genes that has evolved fundamentally to help humans survive and be well.
Previous studies had found that circulating immune cells show a systematic shift in baseline gene-expression profiles during extended periods of stress, threat or uncertainty. Known as conserved transcriptional response to adversity, or CTRA, this shift is characterized by an increased expression of genes involved in inflammation and a decreased expression of genes involved in antiviral responses.
This response, Cole noted, likely evolved to help the immune system counter the changing patterns of microbial threat that were ancestrally associated with changing socio-environmental conditions; these threats included bacterial infection from wounds caused by social conflict and an increased risk of viral infection associated with social contact.
"But in contemporary society and our very different environment, chronic activation by social or symbolic threats can promote inflammation and cause cardiovascular, neurodegenerative and other diseases and can impair resistance to viral infections," said Cole, the senior author of the research.
In the present study, the researchers drew blood samples from 80 healthy adults who were assessed for hedonic and eudaimonic well-being, as well as potentially confounding negative psychological and behavioral factors. The team used the CTRA gene-expression profile to map the potentially distinct biological effects of hedonic and eudaimonic well-being.
And while those with eudaimonic well-being showed favorable gene-expression profiles in their immune cells and those with hedonic well-being showed an adverse gene-expression profile, "people with high levels of hedonic well-being didn't feel any worse than those with high levels of eudaimonic well-being," Cole said. "Both seemed to have the same high levels of positive emotion. However, their genomes were responding very differently even though their emotional states were similarly positive.
"What this study tells us is that doing good and feeling good have very different effects on the human genome, even though they generate similar levels of positive emotion," he said. "Apparently, the human genome is much more sensitive to different ways of achieving happiness than are conscious minds."
###
Other authors on the study included Jesusa M.G. Arevalo and Jeffrey Ma, both of UCLA, and Karen M. Grewen, Kimberly A. Coffey, Sara B. Algoe and Ann M. Firestine of the University of North Carolina.
The research was supported by National Institutes of Health grants R01NR012899, R01CA116778 and P30AG107265.
The UCLA Cousins Center for Psychoneuroimmunology encompasses an interdisciplinary network of scientists working to advance the understanding of psychoneuroimmunology by linking basic and clinical research programs and by translating findings into clinical practice. The center is affiliated with the Semel Institute for Neuroscience and Human Behavior and the David Geffen School of Medicine at UCLA.
For more news, visit the UCLA Newsroom and follow us on Twitter.
[ | E-mail | Share ]
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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Be happy: Your genes may thank you for itPublic release date: 29-Jul-2013 [ | E-mail | Share ]
Contact: Mark Wheeler mwheeler@mednet.ucla.edu 310-794-2265 University of California - Los Angeles
But different types of happiness have different effects, UCLA study shows
A good state of mind that is, your happiness affects your genes, scientists say. In the first study of its kind, researchers from UCLA's Cousins Center for Psychoneuroimmunology and the University of North Carolina examined how positive psychology impacts human gene expression.
What they found is that different types of happiness have surprisingly different effects on the human genome.
People who have high levels of what is known as eudaimonic well-being the kind of happiness that comes from having a deep sense of purpose and meaning in life (think Mother Teresa) showed very favorable gene-expression profiles in their immune cells. They had low levels of inflammatory gene expression and strong expression of antiviral and antibody genes.
However, people who had relatively high levels of hedonic well-being the type of happiness that comes from consummatory self-gratification (think most celebrities) actually showed just the opposite. They had an adverse expression profile involving high inflammation and low antiviral and antibody gene expression.
The report appears in the current online edition of the journal Proceedings of the National Academy of Sciences.
For the last 10 years, Steven Cole, a UCLA professor of medicine and a member of the UCLA Cousins Center, and his colleagues, including first author Barbara L. Fredrickson at the University of North Carolina, have been examining how the human genome responds to stress, misery, fear and all kinds of negative psychology.
In this study, though, the researchers asked how the human genome might respond to positive psychology. Is it just the opposite of stress and misery, or does positive well-being activate a different kind of gene expression program?
The researchers examined the biological implications of both hedonic and eudaimonic well-being through the lens of the human genome, a system of some 21,000 genes that has evolved fundamentally to help humans survive and be well.
Previous studies had found that circulating immune cells show a systematic shift in baseline gene-expression profiles during extended periods of stress, threat or uncertainty. Known as conserved transcriptional response to adversity, or CTRA, this shift is characterized by an increased expression of genes involved in inflammation and a decreased expression of genes involved in antiviral responses.
This response, Cole noted, likely evolved to help the immune system counter the changing patterns of microbial threat that were ancestrally associated with changing socio-environmental conditions; these threats included bacterial infection from wounds caused by social conflict and an increased risk of viral infection associated with social contact.
"But in contemporary society and our very different environment, chronic activation by social or symbolic threats can promote inflammation and cause cardiovascular, neurodegenerative and other diseases and can impair resistance to viral infections," said Cole, the senior author of the research.
In the present study, the researchers drew blood samples from 80 healthy adults who were assessed for hedonic and eudaimonic well-being, as well as potentially confounding negative psychological and behavioral factors. The team used the CTRA gene-expression profile to map the potentially distinct biological effects of hedonic and eudaimonic well-being.
And while those with eudaimonic well-being showed favorable gene-expression profiles in their immune cells and those with hedonic well-being showed an adverse gene-expression profile, "people with high levels of hedonic well-being didn't feel any worse than those with high levels of eudaimonic well-being," Cole said. "Both seemed to have the same high levels of positive emotion. However, their genomes were responding very differently even though their emotional states were similarly positive.
"What this study tells us is that doing good and feeling good have very different effects on the human genome, even though they generate similar levels of positive emotion," he said. "Apparently, the human genome is much more sensitive to different ways of achieving happiness than are conscious minds."
###
Other authors on the study included Jesusa M.G. Arevalo and Jeffrey Ma, both of UCLA, and Karen M. Grewen, Kimberly A. Coffey, Sara B. Algoe and Ann M. Firestine of the University of North Carolina.
The research was supported by National Institutes of Health grants R01NR012899, R01CA116778 and P30AG107265.
The UCLA Cousins Center for Psychoneuroimmunology encompasses an interdisciplinary network of scientists working to advance the understanding of psychoneuroimmunology by linking basic and clinical research programs and by translating findings into clinical practice. The center is affiliated with the Semel Institute for Neuroscience and Human Behavior and the David Geffen School of Medicine at UCLA.
For more news, visit the UCLA Newsroom and follow us on Twitter.
[ | E-mail | Share ]
?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
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Public release date: 29-Jul-2013
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